Cinnamon Bark Essential Oil

[VRI/NE/01-050]

Extraction Method - Steam Distillation

Certificate - ISO, GMP, Organic, IFRA, FSSAI, Halal

Source - Bark

(0)
₹2,655.10

Botanical Name :  Cinnamon zeylanicum Common name:  Cinnamon, Dalchini, Read More

Botanical Name : 

Cinnamon zeylanicum

Common name: 

Cinnamon, Dalchini,

Plant family: 

Lauracae

Genus: 

Cinnamomum

Appearance/Color:

A thin light yellowish brown color and a highly fragrant odor.

Odor:

Oil secures a middle note in aroma. It has a unique fragrance which reminds of the smell of apple sweet straw.

Blends With:

Blended well with cedarwood, frankincense, ylang-ylang, lemon, lavender, neroli and orange to process a good mix of products.

Origin

Madagascar

 

The genus Cinnamomum (Laureaceae family) consists of 250 spices of trees and shrubs distributed in south-east Asia, China, and Australia. It is a small, evergreen tree, 10–15 m tall. The bark is widely employed as a spice, its leaves are ovate-oblong in shape, and 7–18 cm long. The flowers, arranged in panicles, have a greenish color and have a rather disagreeable odor. The fruit is a purple 1 cm berry containing a single seed. It is found in tropical rain forests, where it grows at various altitudes from highland slopes to lowland forests and occurs in both marshy places and on well-drained soils. However, in latitudes with seasonal climatic conditions, they become exceedingly rare. There are two main varieties of cinnamon namely the Ceylon or true cinnamon (Cinnamon zeylanicum) and cassia (Cinnamom aromaticum). C. zeylanicum is native to Sri Lanka and tropical Asia and exotic to several African countries, such as Comoros, Ghana, Madagascar, Mauritius, Nigeria, Seychelles, Sierra Leone, Tanzania, and Uganda.

The most important cinnamon oils in word trade are those from Cinnamomum zeylanicum, Cinnamomum cassia, and Cinnamomum camphora. The cinnamon essential oil composition varies depending on the geographical origin of the spice and the processing conditions. Likewise, it has been detected slight differences between the composition of volatile oils from buds, flowers, and fruits from C. zeylanicum. Jayaprakasha et al. (2000) reported that the oil from buds contains higher amounts of mono and sesquiterpenes, and most characteristic compounds such as cinnamaldehyde are found in flowers and fruits and in lower amounts in buds

 

The barks are commonly used in Cameroon as spices and for the treatment of cardiovascular diseases and cancers. Cinnamaldehyde (1; 75%) and camphor (56%) are the major compounds in oil and stem bark, respectively. Cinnamon extract inhibits the formation of toxic A? oligomers and prevents the toxicity of A? on neuronal PC12 cells. Oral administration of cinnamon extract induced to the reduction of plaques and improvement in cognitive behavior to aggressive Alzheimer's disease transgenic mice. The prevention or treatments of Alzheimer’s disease are mostly based on drugs inhibition of cholinesterase function or formation of amyloid plaque. Pathologies and dementias of the nervous system, such as Alzheimer’s disease and Parkinson’s disease (Lei et al., 2010) can result when tau proteins (tau proteins are proteins that stabilize microtubules) become defective and no longer stabilize microtubules properly. Chemicals able to prevent the tau aggregation may be a key factor in the development of new drugs.

DISCLAIMER

The complete range of conditions or methods of use are beyond our control therefore we do not assume any responsibility and expressly disclaim any liability for any use of this product. Information contained herein is believed to be true and accurate however, all statements or suggestions are made without warranty, expressed or implied, regarding accuracy of the information, the hazards connected with the use of the material or the results to be obtained from the use thereof. Compliance with all applicable federal, state, and local laws and local regulations remains the responsibility of the user.

The FDA has not evaluated the statements on this website. No claims are made by Venkatramna Industries as to the medicinal value of any products from vriaroma.com or by us. The information presented here is for educating our customers about the traditional uses of essential oils and is not intended to diagnose, treat, cure, or prevent any disease. You are responsible for understanding the safe application of these products. If you have any questions, please call or email us for further information.

As per NAHA guidelines, New Directions Aromatics (NDA) does not recommend the ingestion of essential oils. It is imperative to consult a medical practitioner before using Essential Oils for therapeutic purposes. Pregnant and nursing women and those taking prescription drugs are especially advised not to use this product without the medical advice of a physician. The oil should always be stored in an area that is inaccessible to children, especially those under the age of 7.

Cinnamon, the eternal tree of tropical medicine, belongs to the Lauraceae family. Cinnamon is one of the most important spices used daily by people all over the world. Cinnamon primarily contains vital oils and other derivatives, such as cinnamaldehyde, cinnamic acid, and cinnamate. In addition to being an antioxidant, anti-inflammatory, antidiabetic, antimicrobial, anticancer, lipid-lowering, and cardiovascular-disease-lowering compound, cinnamon has also been reported to have activities against neurological disorders, such as Parkinson’s and Alzheimer’s diseases. This review illustrates the pharmacological prospective of cinnamon and its use in daily life.

Cinnamon Bark Essential Oil in Pharma

 The barks are commonly used in Cameroon as spices and for the treatment of cardiovascular diseases and cancers. Cinnamaldehyde (1; 75%) and camphor (56%) are the major compounds in oil and stem bark, respectively. Cinnamon extract inhibits the formation of toxic A? oligomers and prevents the toxicity of A? on neuronal PC12 cells. Oral administration of cinnamon extract induced to the reduction of plaques and improvement in cognitive behavior to aggressive Alzheimer's disease transgenic mice. The prevention or treatments of Alzheimer’s disease are mostly based on drugs inhibition of cholinesterase function or formation of amyloid plaque. Pathologies and dementias of the nervous system, such as Alzheimer’s disease and Parkinson’s disease (Lei et al., 2010) can result when tau proteins (tau proteins are proteins that stabilize microtubules) become defective and no longer stabilize microtubules properly. Chemicals able to prevent the tau aggregation may be a key factor in the development of new drugs.

Essence of Cinnamon Bark Essential Oil

Essential oils of cinnamon bark and leaf are widely used in food flavours, cosmetics and pharmaceuticals. Cinnamon is mainly used in the aroma and essence industries due to its fragrance, which can be incorporated into different varieties of foodstuffs, perfumes, and medicinal products. The Oil is rich in monoterpenoids and phenyl propanoids. (E)-Cinnamaldehyde is the main component of cinnamon bark oil and eugenol the main component of the leaf oil. Benzyl benzoate chemotypes of cinnamon bark and leaf oils and linalool chemotype of cinnamon leaf oil were also reported. Essential oils of other parts such as fruit, fruit stalk, flower, bud, leaf petiole of cinnamon have also reported having medicinal and pharmacological benefits.

COMMON USAGE

·        Stimulates brain function

·        Aids in weight loss

·        Best blood purifier

·        Reduces pain

·        Prevents and controls diabetes

·        Cures different types of infections

·        Possesses quick healing properties

·        Inhibits heart problems

·        Prevents colon cancer

·        Removes bad breath

·        Treats respiratory ailments

Ingredients:

S.No

Key Constituents

Strength (%)

1

Eugenol

2.0-13.3

2

(E)-cinnamyl acetate

0.02-11.6

3

linalool

01.2-6.0

4

(E)-cinnamaldehyde

60.1-78.7

5

p-cymene

1.6-2.4

6

b-caryophyllene

1.3-5.8

7

benzaldehyde

tr-3.2

8

1,8-cineole

0.5-2.3

9

a-terpineol

0.4-1.42

10

b-phellandrene

<2.5

11

Terpinen-4-ol

0.5-1.2

12

Camphor

tr-1.3

13

Benzyl benzoate

tr-01.0

14

Safrole

0-0.05

15

a-caryophyllene

0-01.0

 

TOXICOLOGICAL INFORMATION

Safety Summary

·        Hazardous No Data

·        Contraindications Not Known

Organ Specific Effects

·        Skin corrosion / irritation: May cause an allergic reaction by skin contact.

·        Serious eye damage / irritation: May have reversible effects on the eyes, such as eye irritation which is totally reversible by the end of observation at 21 days.

 

Systemic Effects

·        Acute Toxicity

o   Oral route:DL50 = 2650 mg/kg

o   Dermal route: DL50 > 5000 mg/kg

·        Respiratory sensitization: Not applicable under normal use.

·        Germ cell mutagenicity: Cause for concern owing to the possibility that it may induce heritable mutations in the germ cells of humans.

·        Carcinogenicity: IARH: No component of this product present at levels greater than or equal to 0.1% is identified as probable, possible or confirmed human carcinogen by IARC. ACGIH: No component of this product present at levels greater than or equal to 0.1% is identified as probable, possible or confirmed human carcinogen by IARC.

·        NTP: No component of this product present at levels greater than or equal to 0.1% is identified as probable, possible or confirmed human carcinogen by IARC.

·        OSHA: No component of this product present at levels greater than or equal to 0.1% is identified as probable, possible or confirmed human carcinogen by IARC.

·        Reproductive toxicity: Not specified

·        STOT-single exposure: Not specified

·        STOT-related exposure: Not specified

·        Aspiration hazard: Not specified

 

ECOLOGICAL INFORMATION

·        Toxicity
Harmful to aquatic life with long lasting effects. The product must not be allowed to run into drains or waterways.

·        Substances classified as category 1 Acute toxicity

·        Crustacean toxicity: Duration of exposure : 48 h CE50 = 1.61 mg/l Species: Daphnia sp. OECD Guideline 202 (Daphnia sp. Acute Immobilisation Test)

·        Persistence and degradability Biodegradation is expected

·        Bio-accumulative potential Bioaccumulation is unlikely

·        Mobility in soil Unknown 

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